With Bird flu propaganda everywhere, I thought I’d double-check Professor SARS Ralph Baric’s protocols for fooling around with respiratory viruses and not getting sick. I think he's just evil after all, not suicidal. Interestingly, I found what I believe is good news. Here is my trip down that rabbit hole.
I am not a doctor. This is not PfDA approved. You know the drill.
New Metrics for Evaluating Viral Respiratory Pathogenesis - Ralph S. Baric
In the current work, we utilize infection with mouse adapted Severe Acute Respiratory Syndrome Coronavirus (SARS-CoV) as well as a 2009 Influenza A (IAV) H1N1 isolate (A/California/04/09 (H1N1), henceforth referred to as H1N1-CA04-2009) to model severe acute respiratory disease.
Ralph was working on both SARS (covid) and HIN1 (bird flu) in 2015.
Increasing the intracellular Zn2+ concentration with zinc-ionophores like pyrithione (PT) can efficiently impair the replication of a variety of RNA viruses, including poliovirus and influenza virus. For some viruses this effect has been attributed to interference with viral polyprotein processing.
Positive-stranded RNA (+RNA) viruses include many important pathogens. They have evolved a variety of replication strategies but are unified in the fact that an RNA-dependent RNA polymerase (RdRp) functions as the core enzyme of their RNA- synthesizing machinery. Here, we not only show that corona- and arterivirus replication can be inhibited by increased Zn2+ levels, but also use both isolated replication complexes and purified recombinant RdRps to demonstrate that this effect may be based on direct inhibition of nidovirus RdRps.
Ralph was using zinc and zinc ionophores in 2010 to protect himself but also something I overlooked when I first read this study:
“RNA (+RNA) viruses are unified in the fact that an RNA-dependent RNA polymerase (RdRp) functions as the core enzyme of their RNA- synthesizing machinery. replication can be inhibited by increased Zn2+ levels, but also on direct inhibition of nidovirus RdRps.”
I cheated on my homework here a little. The large language model AI’s are getting vilified many places in the alt-infoverse (WOKE AI!!) but are fiendishly fast at grabbing information when you use specific technical language. I did my own research for several hours and typed the keywords I found into ChatGPT. It came up with most of the same answers I had found but in roughly 20 seconds. It did call me a racist and asked if I wanted a sex change, but other than that… I’m kidding, but I admit having WokeGPT bias and not utilizing what can be a valuable tool with the right words. This isn’t a GPT endorsement, it can certainly be gamed to give false information and there are probably better LLM’s. It is just an observation that if I want to keep up with this never-ending Viral Psyop, I’ve got to research faster, and AI can help. Back to the rabbit hole with a little assistance:
is bird flu a nidovirus?
ChatGPT: bird flu is not a nidovirus. It is caused by influenza viruses, specifically by strains of the Influenza A virus, which belongs to the family Orthomyxoviridae. The term "bird flu" usually refers to avian influenza, which can affect various bird species and, in some cases, spread to humans. These viruses are not related to nidoviruses, which include families like Coronaviridae, Arteriviridae, Roniviridae, and Mesoniviridae.
does Orthomyxoviridae contain RNA-dependent RNA polymerase?
ChatGPT: Yes, Orthomyxoviridae contain RNA-dependent RNA polymerase (RdRp). RdRp is an essential enzyme for the replication and transcription of RNA viruses. In Orthomyxoviridae, this enzyme is responsible for replicating the virus's RNA genome and for synthesizing mRNA, which is used for protein production.
Influenza viruses, the most well-known members of the Orthomyxoviridae family, have segmented RNA genomes. Their RNA-dependent RNA polymerase consists of three subunits: PA, PB1, and PB2. This complex plays a crucial role in both replicating the viral genome and transcribing it into messenger RNA for protein synthesis.
RdRp is a characteristic feature of RNA viruses, allowing them to replicate their RNA-based genomes within a host cell, a key aspect of the viral life cycle.
is ebola a nidovirus?
ChatGPT
No, Ebola is not a nidovirus. Ebola is caused by viruses from the genus Ebolavirus, which belongs to the family Filoviridae. Nidoviruses include families like Coronaviridae, Arteriviridae, Roniviridae, and Mesoniviridae, which have different genetic structures and replication mechanisms compared to filoviruses.
Ebola virus is a negative-sense single-strand RNA virus, whereas most nidoviruses are positive-sense single-strand RNA viruses. Additionally, filoviruses like Ebola are known for their filamentous shape, while nidoviruses often have other structures, such as the crown-like spike proteins characteristic of coronaviruses.
does Filoviridae contain RNA-dependent RNA polymerase?
ChatGPT
Yes, Filoviridae contain RNA-dependent RNA polymerase (RdRp). This enzyme is crucial for the replication and transcription of the viral RNA genome in filoviruses like Ebola and Marburg. RNA-dependent RNA polymerase is responsible for creating copies of the virus's RNA genome, enabling it to multiply within a host cell, and for transcribing the viral genome into messenger RNA (mRNA) to produce viral proteins.
In Filoviridae, RdRp is a component of the viral protein complex associated with the nucleocapsid, and it's essential for the virus's ability to replicate and spread. The enzyme allows the negative-sense RNA genome to be transcribed into positive-sense RNA for protein synthesis and replicated for assembling new virions.
It seems the scary movie viruses Ebola and Marburg have the same Achilles heel as SARS, Influenza and every other RNA virus…RdRP inhibition. The Pfear-mongering over bird flu has the super-scariest of viral properties built into the name (bird). It’s been hammered into our subconscious through tv and movies for as long as there’s been tv and movies.
Speaking of safety, remember Ralph Baric and the like already have and use simple defenses against their new viral toys, or they would not be playing with them. As we learned in the movie ‘Outbreak’ above, those bio-suits aren’t 100% reliable. I don’t know precisely they use (besides zinc), but I do know they don’t fear the viruses they tell us to fear.
In the virus drug game, they hunt for natural anti-viral substances (which they can’t patent), change it by adding something synthetic (which they can patent) make the drug and sell it. It seems that’s exactly what is happening with the RdRp inhibtors:
Inhibition of Influenza Virus Polymerase by Interfering with Its Protein-Protein Interactions
One of the most promising targets for the development of new antiflu therapeutics is the viral RNA-dependent RNA polymerase (RdRP).
An overview of small molecules able to interfere with flu RdRP functions exploiting the PPI inhibition approach is described. In particular, an update on the most recent inhibitors targeting the well-consolidated RdRP PA-PB1 subunit heterodimerization is mainly reported, together with pioneer inhibitors targeting other virus-virus or virus-host interactions involving RdRP subunits. As demonstrated by the PA-PB1 interaction inhibitors discussed herein, the inhibition of flu RdRP functions by PPI disrupters clearly represents a valid means to identify compounds endowed with a broad spectrum of action and a reduced propensity to develop drug resistance, which are the main issues of antiviral drugs.
Blocking nuclear localization of flu RdRP subunits is a potentially promising mechanism for new antivirals
This article provides an overview of RdRp inhibitors that have been launched or are in clinical studies for the treatment of RNA virus infections, including COVID-19.
RdRp inhibiting plants is where they are looking for new drug ideas, there are many such studies underway. Back down the rabbit hole:
list ten plant extracts that are RNA-dependent RNA polymerase inhibitors
ChatGPT
RNA-dependent RNA polymerase (RdRp) inhibitors are substances that can block the function of the enzyme responsible for replicating RNA in RNA viruses. Plant extracts can contain various compounds with antiviral properties, including those that can inhibit RdRp. Here is a list of ten plant extracts or derived compounds that have shown RNA-dependent RNA polymerase inhibitory activity:
Glycyrrhizin: An active component from licorice root (Glycyrrhiza glabra), shown to have antiviral effects against several RNA viruses, including SARS-related coronaviruses.
Andrographolide: Derived from Andrographis paniculata, it has exhibited antiviral activity, including potential inhibition of RdRp.
Quercetin: A flavonoid found in many plants and foods like onions, apples, and berries, with potential RdRp inhibitory activity.
Catechins: Found in green tea (Camellia sinensis), they have shown antiviral properties, including potential inhibition of RNA-dependent RNA polymerase.
Emodin: An anthraquinone from rhubarb (Rheum spp.) and other plants, known to have antiviral effects.
Curcumin: The active component of turmeric (Curcuma longa), which has been studied for its broad-spectrum antiviral activity, including inhibition of viral replication.
Resveratrol: A polyphenol found in grapes and red wine, with studies suggesting antiviral activity through various mechanisms, including potential RdRp inhibition.
Apigenin: A flavonoid present in many plants like chamomile (Matricaria chamomilla) and parsley, with demonstrated antiviral properties.
Berberine: An alkaloid found in plants like Berberis and Coptis, with antiviral activities and potential RNA polymerase inhibition.
Echinacea purpurea: Extracts from this plant are known for their immune-boosting and antiviral properties, with studies suggesting possible inhibition of viral replication, though the specific mechanisms, including RdRp inhibition, are still under investigation.
These plant extracts and compounds have shown various degrees of effectiveness in inhibiting RNA viruses, often with a focus on other mechanisms like immune modulation. The extent to which they specifically inhibit RNA-dependent RNA polymerase can vary, and more research is typically needed to confirm the exact mode of action for each.
Interesting. I recognize six of those ten from existing alternative Covid protocols.
list ten plant being investigated as RNA-dependent RNA polymerase inhibitors
ChatGPT: Research on plant-based inhibitors for RNA-dependent RNA polymerase (RdRp) is growing, with several plant extracts and derived compounds demonstrating potential antiviral properties. While there's active investigation in this area, here's a list of ten plants that are among those being studied for their potential as RdRp inhibitors in RNA viruses:
Glycyrrhiza glabra (Licorice): Glycyrrhizin, a compound derived from licorice, has shown antiviral activity and is being investigated for its potential to inhibit RdRp.
Andrographis paniculata: This plant contains andrographolide, which has been studied for its antiviral effects and potential role in inhibiting RdRp.
Camellia sinensis (Green Tea): Catechins from green tea are known for their antiviral properties, and researchers are exploring their ability to inhibit RNA polymerase.
Curcuma longa (Turmeric): Curcumin, derived from turmeric, is under investigation for its broad-spectrum antiviral activities, including effects on RNA viruses.
Emblica officinalis (Indian Gooseberry): This plant is rich in antioxidants and has been studied for its potential antiviral properties, with interest in its impact on RdRp.
Phyllanthus niruri: Known for its traditional medicinal uses, this plant contains compounds that are being researched for antiviral activity, including possible RdRp inhibition.
Berberis vulgaris (Barberry): Berberine, an alkaloid found in barberry, is being studied for its antiviral properties and potential role in inhibiting viral replication.
Salvia officinalis (Sage): Known for its antimicrobial properties, sage is also being investigated for antiviral effects and its impact on RNA viruses.
Withania somnifera (Ashwagandha): This adaptogenic plant is under research for its antiviral potential, including possible effects on RdRp.
Ocimum sanctum (Holy Basil): Known for its wide-ranging health benefits, holy basil is also being studied for its antiviral properties and potential impact on RNA polymerase.
I believe humans were designed to live on this planet as was everything else. Knowledge of diet and specific plants to treat illness was built up over eons, yet somehow it was lost to the majority of people. Occulted, you might say. It still exists though, and people are remembering and re-learning. I’ve linked two of my favorite protocols below, for anyone to do their own research on and help escape the fear matrix they use to control us. They can’t use viruses to scare us into a dystopian medical tyranny if no one is afraid of viruses.
Early Covid-19 Treatment Guide (worldcouncilforhealth.org)
Treatment Protocols - FLCCC Alliance (covid19criticalcare.com)
Excellent information, Rob! I regret not knowing about your Substack until today. I will be sharing And encouraging others to follow you!